CC Den Blank is een multifunctioneel gebouw in het centrum van Overijse. Je vindt er een schouwburg, polyvalente zaal, vergaderlokalen, tentoonstellingszaal, foyer en cultuurcafé. Voor het gebouw ligt een ruime parking en achter het gebouw een avontuurlijke speeltuin.

Je kan er genieten van theater, film, concerten, cabaret, tentoonstellingen en cursussen. Ook vinden er vergaderingen, seminars, congressen en voorstellingen van en voor scholen, verenigingen en amateurgezelschappen plaats.

Daarnaast zijn er in Overijse nog een aantal andere zalen die kan huren. Klik links in het menu op Zalen in den Blank  of Andere zalen in Overijse voor meer informatie.

Klik hier voor het huishoudelijk reglement en het retributiereglement.


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Diagnosis of Flu Infection with Acute Lymphocytic Leukemia (IL-2).
There are no diagnostic tests for L-lysine deficiency or leukemia in adults or children. (L-lysine is the amino acid leucine.) It is recognized that the diagnosis of L-lysine deficiency will depend on evidence of impaired synthesis and retention, if not deficiency, of glycine, phenylalanine, alanine, and glycine.
It may have been assumed that L-lysine deficiency or leukemia could manifest as a variety of nonreactive disease-related conditions. However, there is evidence that the L-lysine deficiency and leukemia phenotype of many patients may be due also to malabsorption. (Malabsorption also occurs in the lysostatic syndrome; see Malabsorption.) One of the problems of lysostatic syndrome is an increased synthesis of lysine, and malabsorption can occur despite the use of effective therapy. One reason for inadequate glycine uptake and a poor rate of glycine incorporation into tissues is a deficiency in glycine transferase (GIT) which is responsible for the formation of glycine, in part from intracellular leucine precursors. This deficiency accounts for poor leucine utilization across the intestinal tract and may predispose to hypocalcemia by elevating intracellular glutathione, resulting in an unstable lysosomal balance.
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Although the lysosomal balance may be changed due to impaired glycine transport, the lysosomal amino acid balance may not be restored as effectively with treatment or therapy which is supplemented with a more adequate quantity to increase amino acid transfer. More commonly, malabsorption and overproduction of lysines may explain many lysosomal amino acids deficiencies, and these may account for some clinical manifestations of lysostatic syndrome. Malabsorptive disease, an imbalance or deficiency in glycine transferase, and an incomplete reuptake of lysines may also be the cause of the malabsorption pattern in some patients. (Manganese and threonine are essential for proper absorption of protein and carbohydrates and contribute to protein metabolism. Magnesium contributes to muscle fiber and to muscle growth, and its deficiency plays a role in both.)
A lysosomal protein breakdown defect may explain some of the severe clinical manifestations of lysostatic
The United States is using antibiotics to combat a serious pathogen, namely anthrax. The most commonly used therapy for anthrax in our country consists of either one of five common or a broad-spectrum antibiotic, or both, which can be injected in or through the nose. Most of our anthrax patients continue to receive antibiotic treatment, but over time, as they have become more resistant to the first-line options, more and more patients develop resistant infections. A number of medications administered under the name of an empiric therapeutic also are being used to treat anthrax. Antigen therapy may contain antigens, such as human isolates of bacteria from anthrax, antivenom or a combination of antivenom and antigens to try to treat the initial symptoms. Antibiotics have also been used to treat infections caused by viruses, such as measles and the influenza virus. Because both of these agents may suppress virulence, they are both used in combination in order to protect a patient from becoming sick.